News | Air Pollution Linked to Male Infertility Risk, Noise May Affect Female Fertility



News | Air Pollution Linked to Male Infertility Risk, Noise May Affect Female Fertility


A recent study published in The BMJ found that long-term exposure to traffic noise and air pollution may be closely associated with higher infertility risk in men and women. It was the first nationwide study to assess the effects of both air pollution and traffic noise on infertility in both sexes.


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Infertility Risk Factors

Infertility is defined as failure to become pregnant after one year of regular sex without contraception. This global health issue affects physical health and is also associated with prolonged stress, anxiety, and depression. In addition to age, tobacco and alcohol use, chronic disease, and obesity, air and noise pollution are increasingly recognized as possible risk factors.


Previous studies have linked air pollution to lower sperm count, motility, and normal morphology in men, as well as lower fertility-treatment success rates in women. Traffic noise may affect reproductive function by triggering stress responses and disrupting sleep, although research on noise and infertility remains limited.


Methods and Results

Conducted in Denmark, the study analyzed data from more than 900,000 men and women ages 30 to 45 and assessed long-term exposure to particulate matter smaller than 2.5 micrometers (PM2.5) and road traffic noise. The Danish national patient registry provided individual data on pollution and noise exposure and socioeconomic variables.


Long-term PM2.5 exposure was associated with higher male infertility risk, especially among men ages 37 to 45. Long-term road traffic noise exposure was associated with significantly higher infertility risk in women over 35, particularly ovulatory disorders and unexplained infertility. Associations with noise were weaker in younger women and in men.


Conclusion

The study found a significant association between air pollution and male infertility and a higher infertility risk among women over 35 exposed to long-term traffic noise. Although it cannot establish causation, the findings may inform air-pollution and noise-control policies intended to improve fertility rates in Western countries.


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